Previous studies have suggested that diabetes mellitus (DM) is an outcome of exposure to air pollution, and metabolic detoxification genes affect air pollution–related outcomes.
 The researchers evaluated associations between air pollutants and markers of insulin resistance (IR), an underlying mechanism of type 2 DM, and effect modification by GSTM1, GSTT1, and GSTP1 genotypes among elderly participants in the Korean Elderly Environmental Panel (KEEP) study.


Human exposure to particulate matter (PM) was determined in Urban Environment of Delhi, India. Samples were collected using eight stages impactor (Marple Cascade Impactor) at five sites during August to October 2011. Three samples were collected from each site; samples were analyzed in eight size ranges gravimetrically and chemically (for metals). Maximum (985 μg m-3) exposure to PM was in Okhala an industrial site and minimum (404 μg m-3) in Jawaharlal Nehru University, an educational Institute.

As India's capital, Delhi has grown across sectors - industry, transport, and housing - which contribute to an increase in air pollution. This, in turn, has increased health risks, which are refl ected in a rise in respiratory ailments. While the benefi ts of some interventions in the transport sector have been apparent, it is time to focus on low-hanging fruit in other sectors in order to improve air quality and public health in the city.

Street-level concentrations of nitrogen dioxide (NO2) and particulate matter (PM) exceed public health standards in many cities, causing increased mortality and morbidity. Concentrations can be reduced by controlling emissions, increasing dispersion, or increasing deposition rates, but little attention has been paid to the latter as a pollution control method. Both NO2 and PM are deposited onto surfaces at rates that vary according to the nature of the surface; deposition rates to vegetation are much higher than those to hard, built surfaces.